From: Subject: =?iso-8859-1?Q?Superbugs:_Not?= =?iso-8859-1?Q?_super_after_a?= =?iso-8859-1?Q?ll?= Date: Tue, 20 Jan 2004 20:34:45 -0500 MIME-Version: 1.0 Content-Type: multipart/related; boundary="----=_NextPart_000_0052_01C3DF94.D7A78620"; type="text/html" X-MimeOLE: Produced By Microsoft MimeOLE V5.00.3314.1001 This is a multi-part message in MIME format. ------=_NextPart_000_0052_01C3DF94.D7A78620 Content-Type: text/html; charset="iso-8859-1" Content-Transfer-Encoding: quoted-printable Content-Location: =?iso-8859-1?B?bWh0bWw6ZmlsZTovLy8=?= =?iso-8859-1?B?ZXhwb3J0L2hvbWUvY2g=?= =?iso-8859-1?B?ZXVuZy9CaWJsZS9Fdm8=?= =?iso-8859-1?B?bHV0aW9uL0NvbnMvU3U=?= =?iso-8859-1?B?cGVyYnVncy1Ob3Qtc3U=?= =?iso-8859-1?B?cGVyLWFmdGVyLWFsbC4=?= =?iso-8859-1?B?bWh0?= Superbugs: Not super after all

Defending the Christian faith beginning with = Genesis

Superbugs: Not super after all

Why drug-resistant germs in hospitals = don't show=20 that Darwin was right

by = Carl Wieland

First published in:
Creat= ion=20 Ex Nihilo 20(1):10-13, Dec. 1997=96Feb. 1998

AFTER over 12 years as a medical practitioner, I = suddenly=20 found myself an avid consumer, rather than a provider, of medical = care.=20 Involved in a serious road accident in 1986, I spent many months = in=20 hospital, including weeks in an intensive care unit.

While in intensive care, I became infected with = one of the=20 varieties of so-called supergerms, which are the scourge of = modern=20 hospitals. These are strains of bacteria which are resistant to = almost=20 every (and in some cases every) type of antibiotic known to = man.

Several others in the same unit with me died as a = result of=20 infection by the same bacterial strain. The germs overwhelmed = their immune=20 systems and invaded their bloodstream, untouched by the most = expensive and=20 sophisticated antibiotics available.

This supergerm problem¹= =20 is an increasingly serious concern in Western countries. It = strikes=20 precisely those hospitals which are more 'high-tech', and handle = more=20 serious illnesses. Applying more disinfectant is not the answer; = some=20 strains of germs have actually been found thriving in bottles of = hospital=20 disinfectant! The more antibacterial chemical 'weapons' are being = used,=20 the more bacteria are becoming resistant to them.

The reality of increasing bacterial resistance = seems at=20 first to be an obvious example of onwards and upwards evolution. = But the=20 facts, when carefully examined, show otherwise.

NATURAL SELECTION, BUT NOT EVOLUTION

Evolution is basically the belief that everything = has made=20 itself=97that natural processes (over millions of years, without = miraculous,=20 divine input of intelligence) have created an increasingly complex = array=20 of creatures. According to evolution, there was once a time when = none of=20 the creatures in the world had lungs. This means that there was no = genetic=20 information (the 'blueprint' for living things, carried on the = molecule=20 DNA) for lungs=97anywhere. Then, at a later time, 'lung = information' arose=20 and was added to the world, but no 'feather information' as = yet=97feathers=20 evolved later.

Keep = up to date=20 with the latest creation information!

Creation=20 Magazine

Keep your family informed = on the=20 latest easy-to-understand evidences for creation and against = evolution! This unique full-color family magazine gives God = the=20 glory, refutes evolution, and gives you the answers to = defend your=20 faith. Exciting articles and great witnessing material you = won't=20 find anywhere else! Includes a beautifully illustrated = full-color=20 children=92s section in every issue. Powerful ammunition to=20 intelligently discuss nature, history, science, the Bible, = and=20 related subjects. Delivered to your home every three=20 months!

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In other=20 words, for every feature which arises by evolution, there would = need to be=20 new genetic information added to the total information in the = biosphere=20 (i.e., all the information in all creatures on earth). Some = features could=20 be lost subsequently, of course, so there will not always be a = gain, but=20 if microbes turned into magpies, maple trees and musicians, there = must=20 have been a massive net increase in information. This is = not just=20 any jumble of chemical sequences, but meaningful information, = since it=20 codes for complex structures which have purposeful functions.

So if new information, new functional complexity, = can be=20 shown to be arising by itself where previously there was none, = this would=20 give some credibility to the idea of molecules-to-man evolution, = although=20 it would not strictly prove that it had occurred.

However, it can be shown that in every situation = where=20 populations of living things change, they do so without increase = (and=20 often with a decrease) of information. Thus, it is completely = illegitimate=20 for anyone to claim that such changes show 'evolution = happening'.

Let's look at what is known about how the = superbugs=20 became resistant, and ask=97did any new structures or functions = arise in the=20 process (which is another way of asking whether there was any = evidence of=20 evolution)?

There are a number of different ways in which = germs can=20 become resistant to these poisons. A 'superbug' is, by definition, = resistant to many different antibiotics. It may have become = resistant to=20 antibiotic A in one way, to antibiotic B in a completely different = way,=20 and to antibiotic C in another way again. So if we look at = all the=20 known ways of resistance arising in a population of germs, we will = see if=20 any of them are uphill, information-adding processes.

1. Some germs already had the = resistance.

If out of a million bacteria, five already have a = feature=20 that makes them resistant (however that arose) to, say, = penicillin, then=20 soaking them in penicillin will kill all of them except for the = five. Now=20 the body's natural defences will often 'mop up' such a small = population=20 before it can multiply and cause harm, so resistance will not = become a=20 problem. However, if that doesn't happen, then those five germs = can=20 multiply, and their offspring will obviously also be resistant. So = within=20 a short time, there will be millions of germs resistant to = penicillin.=20 Notice that:

This is why multiple resistance to major = antibiotics is=20 more common in hospitals which treat more serious conditions=97 = these are=20 the hospitals which will frequently be using the sophisticated,=20 expensive 'heavy artillery' antibiotics, so this sort of = 'natural=20 selection' will happen more often.
In this kind of instance, the information to = resist the=20 antibiotic was already there in the bacterial population=97it = did not=20 arise by itself, or in response to the antibiotic.

That some germs were already resistant to man-made = antibiotics before these were invented is common knowledge to=20 microbiologists. Soil samples from villages where modern = antibiotics had=20 never been used show that some of the germs are already resistant = to drugs=20 like methicillin which have never existed in nature. Bacteria = revived from=20 the frozen intestines of explorers who died in polar expeditions = carried=20 resistance to several modern antibiotics, which had not been = invented when=20 the explorers died.²=

2. Some germs directly transfer their = resistance to=20 others.

In an amazing process, the closest thing to sex in = bacteria,=20 one germ inserts a tiny tube into another, and a little loop of = DNA called=20 a 'plasmid' transfers from one to another. This sort of gene = transfer,=20 which can obviously pass on information for resistance to a drug, = can even=20 happen between different species of bacteria.

Notice, again, that the information for the = resistance must=20 already exist in nature before it can be passed on. There is no = evidence=20 of anything totally new arising that was not there before. This is = information transfer, not information creation.

So far, we have dealt with situations in which = resistance=20 was obviously already there. Evolutionists would claim, of course, = that=20 such resistance evolved originally in the (unobservable) past. = However, if=20 observed changes in the present do not show us new information, = what=20 support is there for the idea that such information arose in the = past? The=20 mechanism that is put forward for this past evolution is = invariably=20 mutation=97a copying mistake, an accidental change in the DNA code = passed on=20 to the offspring. So that brings us to the final way in which = bacteria can=20 become resistant.

3. Some germs become resistant through = mutation.

Interestingly, where this happens, there is no = clear-cut=20 evidence of information arising. All such mutations appear to be=20 losses of information, degenerative changes. For example, = loss of a=20 control gene may enhance resistance to penicillin.³=

Some antibiotics need to be taken into the = bacterium to do=20 their work. There are sophisticated chemical pumps in bacteria = which can=20 actively pump nutrients from the outside through the cell wall = into the=20 germ's interior. Those germs which do this efficiently, when in = the=20 presence of one of these antibiotics, will therefore efficiently = pump into=20 themselves their own executioner.

However, what if one of these bacteria inherits a = defective=20 gene, by way of a DNA copying mistake (mutation) which will = interfere with=20 the efficiency of this chemical pumping mechanism? Although this = bacterium=20 will not be as good at surviving in normal circumstances, this = defect=20 actually gives it a survival advantage in the presence of the = man-made=20 poison.⁴= =20 Once again, we see that information has been lost/corrupted, not = gained.=20

SUPERWIMPS

It is precisely because the mutations which give = rise to=20 resistance are in some form or another defects, that so-called = supergerms=20 are not really super at all=97they are actually rather = 'wimpy'=20 compared to their close cousins. When I was finally discharged = from=20 hospital, I still had a strain of supergerm colonizing my body. = Nothing=20 had been able to get rid of it, after months in hospital. However, = I was=20 told that all I had to do on going home was to 'get outdoors a = lot,=20 occasionally even roll in the dirt, and wait.' In less than two = weeks of=20 this advice, the supergerms were gone. Why? The reason is that = supergerms=20 are actually defective in other ways, as explained. Therefore, = when they=20 are forced to compete with the ordinary bacteria which normally = thrive on=20 our skin, they do not have a chance. They thrive in hospital = because all=20 the antibiotics and antiseptics being used there keep wiping out = the=20 ordinary bacteria which would normally out-compete, wipe out and = otherwise=20 keep in check these 'superwimps'.⁵=

If they are 'weaker', then why do they cause so = much death=20 and misery in hospitals? These bacteria are not more = aggressive=20 than their colleagues, it is only that doctors have less power to = stop=20 them. Also, those environments which will tend to 'select' such = resistant=20 germs, like intensive care units, are precisely the places where = there=20 will be critically injured people, physically weakened and often = with open=20 wounds.

This is why more than one microbiologist concerned = about=20 these super-infections has mused (only partly tongue in cheek) = that the=20 best thing to happen in major hospitals might be to dump = truckloads of=20 germ-laden dirt into the corridors, rather than keep on applying = more and=20 more chemicals in a never-ending 'arms race' against the bacteria. = In=20 other words, stop using the antibiotics (which of course is hardly = feasible), and all this 'evolution' will reverse itself, as the = bacterial=20 populations shift back again to favour the more hardy, less = resistant=20 varieties.

SUMMARY AND CONCLUSION

Supergerms are actually not super = at all.=20 They are generally less hardy, and less fit to survive outside = of the=20 special conditions in hospitals.=20
There are many instances in which germs become = resistant=20 by simple selection of resistance which already existed = (including that=20 'imported' from other bacteria).=20
Where a mutational defect causes resistance, the = survival=20 advantage is almost always caused by a loss of information. In = no case=20 is there any evidence of an information-adding, 'uphill' change. =
Supergerms give no evidence to sustain = the claim=20 that living things evolved from simple to complex, by adding = information=20 progressively over millions of years.=20

POSTSCRIPT

Death, suffering and disease (including infection) = are part=20 of the curse which came upon a once-perfect world through the = rebellion of=20 our original ancestor, Adam, against his Maker.

Bacteria actually provide evidence against = evolution.=20 Bacterial populations multiply at incredibly high rates. In only a = matter=20 of a few years, bacteria can go through a massive number of = generations,=20 equivalent to millions of years in human terms. Therefore, since = we see=20 mutation and natural selection in bacterial populations happening = all the=20 time, we should see tremendous amounts of real evolution = happening.=20 However, the bacteria we have with us today are essentially the = same as=20 those described by Robert Koch a century ago. In fact, there are = bacteria=20 found fossilised in rock layers, claimed by evolutionists to be = millions=20 of years old, which as far as one can tell are the same as = bacteria living=20 today.

The famous French biologist Pi=E8rre Grass=E9, who = held the=20 chair of evolution at the Sorbonne for many years, admitted that = mutations=20 in bacteria simply showed shifts back and forth around a mean, but = no net=20 effect. Overall, he said, 'mutations do not produce any kind of=20 evolution.'⁶=

When next you read about supergerms, = remember that=20 everything known about them is consistent with the Genesis = creation of an=20 originally good, complex world ruined by sin.

REFERENCES AND NOTES

Common types of bacteria to = become=20 resistant to many different types of antibiotics at once (called = multiple drug resistance) areKlebsiella,=20 Pneumococcus, and Staphylococcus. The term 'golden = staph'=20 has become a lay expression for these superbugs, but it is = actually a=20 correct lay term for the most common type of staph, otherwise = known as=20 S. aureus or S. pyogenes, which applies even if = the bug is=20 not multiply resistant. Return = to=20 text.
R. McGuire, 'Eerie: Human = Arctic fossils=20 yield resistant bacteria'Medical Tribune = December 29,=20 1988, pp. 1, 23. Return = to=20 text.
The enzyme penicillinase, = produced by some=20 bacteria, destroys penicillin. If a member of a bacterial strain = producing a modest amount of this substance were to inherit a = mutational=20 defect which damaged or deleted the gene controlling production = of this=20 enzyme, the organism would invest a lot of resources into = producing=20 copious amounts of penicillinase. Thus, this defect would be an=20 advantage in an environment containing penicillin, but would be = a=20 disadvantage otherwise. Once again, a loss is involved. There is = no=20 evidence that the complex information coding for penicillinase=20 production arose by mutation. Return = to=20 text.
For a somewhat more detailed = and technical=20 treatment of the whole matter of antibiotic resistance, with = further=20 references, see also C. Wieland, 'Antibiotic Resistance in=20 Bacteria',CEN Technical Journal = 8(1):5=966,=20 1994. Return=20 to text.
This of course is a real = dilemma facing the=20 medical profession, especially when faced with patient demand = for=20 antibiotics for illnesses which would probably get better = without them.=20 The more that antibiotics are used, the less effective they = become for=20 some of these life-threatening conditions. Return = to=20 text.
P-P Grass=E9,⁼Evolution of=20 Living Organisms, Academic Press, New York, 1977, p. 88. Return = to=20 text.

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